章 急性胰腺炎与全身性炎症反应综合征
1.Deoxycholic acid inhibited proliferation and induced apoptosis and necrosis byregulating the activity of transcription factors in rat
pancreatic acinar cell lineAR42J(脱氧胆酸通过调节转录因子的活性抑制大鼠胰腺腺泡细胞系AR42J的增殖并诱导其凋亡和坏死)
2.Regulatory effects of apoptosis-associated speck-like protein on cytokines in theP388D1 macrophage-like cell line(凋亡相关斑点样蛋白对 P388D1巨噬细胞样细胞系细胞因子的调控作用)
3.CEBPE Promotes LPS-Induced IL-1B Transcription and Secretion in AlveolarMacrophages via NOD2 Signaling (CEBPB促进LPS诱导的肺泡巨噬细胞IL-1p的转录和分泌)
4.Identification of Key Biomarkers Associated with Immunogenic Cell Death andTheir Regulatory Mechanisms in Severe Acute
Pancreatitis Based on WGCNA andMachine Learning(基于WGCNA 和机器学习方法对重症急性胰腺炎时免疫源性细胞死亡及
其调节机制相关的关键生物标志物的确定)
5.Exosome-derived CIRP: An amplifier of inflammatory diseases(外泌体特异性CIRP;炎症反应的放大器)
6.Protective Effects of Emodin-Induced Neutrophil Apoptosis via the Ca*-Caspase12 Pathway against SIRS in Rats with Severe
Acute Pancreatitis (大黄素诱导的中性粒细胞凋亡通过Ca*-Caspase 12途径对重症急性胰腺炎大鼠的SIRS 具有保护作用)
7.Inhibition of Necroptosis in Acute Pancreatitis: Screening for RIPK1 Inhibitors(急胰腺炎坏死性凋亡的抑制:RIPK1抑制剂的筛选)
8.Regulatory effects of emodin on NF-kB activation and infammatory cytokine expressiorin RAW 264.7 macrophages (大黄素对
RAW 264.7 巨噬细胞中NF-xB活化和炎症因子表达的调节作用)
9.Qingyi Deces acute biliary pancreatitis by targeting Gpbar1NF-rb pathway (清胰汤靶向Gpbar1NF-rb通路缓解急性胆源性胰腺炎)
10.Effects of Qingyi decoction onammatory markers in patients with acute pancreatitis: Aeta-analysis (清胰汤对急性胰腺炎患者炎
性标志物的影响:芸萃分析)
第二章 急性胰腺炎肠道屏障损伤
1.Intestinal barrier damage, systemic inflammatory response syndrome, and acute lung in-jury: A troublesome trio for acute
pancreatitis(肠道屏障损伤、全身性炎症反应综合征和肺损伤:急性胰腺炎的三重奏)
2.The gut-lung axis in severe acute Pancreatitis-associated lung injury: The protection bythe gut microbiota through short-chain
fatty acids(肠道菌群通过短链脂肪酸对急性胰腺炎肺损伤时肠-肺轴的保护作用)
3.Intestinal Microbiota - An Unmissable Bridge to Severe Acute Pancreatitis-AssociatedAcute Lung Injury (肠道菌群对急性胰腺炎
相关性肺损伤具有不可或缺的作用)
4.Therapeutic effect of Qingyi decoction in severe acute pancreatitis-induced intestinal
bar-rier injury(清胰汤对重症急性胰腺炎所致肠道屏障损伤的治疗作用)
5.Qingyi decoction attenuates intestinal epithelial cell injury via the calcineurinnuclearfactor of activated T-cells pathway(清胰汤
通过NFAT 细胞通路减缓肠上皮细胞损伤)
第三章 急性胰腺炎肺损伤
1.Effects and mechanisms of alveolar type Il epithelial cell apoptosis in severe pancreatitis-induced acute lung injury (肺泡I型上皮细
胞凋亡在重症急性胰腺炎所致急性肺损伤中的作用和机制)
2.Enhancement of ICAM-I via the JAK2STAT3 signaling pathway in a rat model of severeacute pancreatitis-associated lung injury
(JAK2STAT3 信号通路在重症急性胰腺炎相关肺损伤大鼠模型中对ICAM-I的增强效应)
3.Inhibition of SOCs Attenuates Acute Lung Injury Induced by Severe Acute Pancreatitis inRats and PMVECs Injury Induced by
Lipopolysaccharide(抑制钙库操纵性钙离子通道可减轻大鼠重症急性胰腺炎肺损伤和脂多糖所致肺血管内皮细胞损伤)
Effect of angiopoietin-like protein 4 on rat pulmonary microvascular endothelial cells ex-posed to LPS(血管生成素样蛋白4对 LPS
所致大鼠肺微血管内皮细胞的影响)
4.Effects of hepoxia-inlucible factor-lor andatrix metalloproteinase-9 on alveolar-capillary barrier disru
ung edemaodels of severe acute pancreatitis associated lung injury (缺氧诱导因子-1c和基质金属蛋白酶-9对重症急性胰腺炎相关肺损伤大鼠肺泡-毛细血管屏障破坏和
肺水肿的影响)
6.Multifunctional neuron-specific enolase: its ro
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